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Novedades Junio 2026

Conclusión: Elevated Lp(a) is associated with increased coronary artery disease severity and higher mid-term MACE risk after AMI.

JAAPA. 2026 Jun 1;39(6):16-21. doi: 10.1097/01.JAA.0000000000000361. Epub 2026 May 26.

Autores:

Nolan Crain  1

Conclusión: In our population, Lp(a) levels were not associated with either office or out-of-office BP values, irrespective of antihypertensive treatment status. The role of Lp(a) in hypertension warrants further investigation.

Conclusión: CAD PRS, LDL-C, hsCRP, and Lp(a) show independent age- and sex-specific associations with CAD. Measuring all 4 biomarkers may improve midlife CAD risk prediction for both male and female patients.

Conclusión: Elevated Lp(a) is associated with higher relative risk across CAC strata, including CAC of 0. Among individuals with CAC of 0, absolute event rates remain low even when Lp(a) is elevated. CAC scoring remains a powerful tool for risk assessment among individuals with elevated Lp(a).

BMC Cardiovasc Disord. 2026 Apr 16;26(1):460. doi: 10.1186/s12872-026-05847-0.

Autores:

Tao Sun  1 , Wenhao Zhang  2 , Pingyan Fei  1 , Yueping Shi  1 , Xuelian Zhang  3

Conclusión: When HCY is elevated, patients with increased Lp(a) experience amplified risk of recurrent cardiovascular events. This association shifts when HCY is at low levels. Future efforts should emphasize combined assessment of Lp(a) and HCY and explore targeted intervention strategies to reduce residual cardiovascular risk.

Prog Lipid Res. 2026 Jun:101:101375. doi: 10.1016/j.plipres.2025.101375. Epub 2025 Dec 24.

Autores:

Ivan Antipenko  1 , Anna Stepanova  2 , Maxim Shkurnikov  3 , Kianoush Jeiran  4 , Ancha Baranova  5 , Alexander Tonevitsky  6

Curr Atheroscler Rep. 2026 Jun 3;28(1):59. doi: 10.1007/s11883-026-01419-x.

Autores:

Khalil Anchouche  1 , Nicholas Koran  2 , George Thanassoulis  3   4

Diabetes Metab Syndr. 2026 Apr;20(4):103422. doi: 10.1016/j.dsx.2026.103422. Epub 2026 Apr 25.

Autores:

Sudipta Chattopadhyay  1 , Jason Sangha  2 , Thozhukat Sathyapalan  3

Conclusión: Lp(a) is inversely related to T2DM risk only in subjects with severe prediabetes. Direct association between Lp(a) and cardiovascular outcomes is seen only in subjects without prediabetes.

J Clin Lipidol. 2026 Jun;20(6):1054-1064. doi: 10.1016/j.jacl.2026.03.019. Epub 2026 Apr 1.

Autores:

Martin D Mulligan  1 , Rahul S Gandhi  2 , Rishabh Vishwakarma  3 , Romit Bhattacharya  4

Conclusión: No statistically significant differences in Lp(a) reduction were observed between currently available PCSK9-targeting medications. Agent selection may reasonably be based on non-efficacy factors, including administration frequency, cost, and patient preference.

Cardiovasc J Afr. 2024 Nov-Dec;35(4):246-250. doi: 10.5830/CVJA-2023-038. Epub 2023 Aug 25.

Autores:

Songül Usalp  1 , Emine Altuntaş  2 , Bayram Bağırtan  2 , Kanber Ö Karabay  2

Conclusión: Lp(a) level was found to be a higher and a possible independent risk factor in young patients who presented with STEMI for the first time, compared to the middle-aged patient group. Lp(a) is a highly atherogenic molecule and it has been associated with stroke, heart failure, aortic stenosis, as well as coronary artery disease. Measurement of Lp(a) levels may be recommended in young patients with high cardiovascular risk.

Conclusión: In patients with elevated Lp(a), GLP-1RA therapy was associated with substantial reductions in mortality and cardiovascular events. These findings suggest potential cardioprotective effects of GLP-1RAs in a high-risk population with limited therapeutic options and support further prospective evaluation.

Conclusión: Cascade screening for elevated Lp(a) in relatives of patients with CAD is feasible in a real-world setting. Screening was up to five times more effective when initiated from patients with elevated Lp(a) than with low.

Conclusión: In this large primary prevention cohort, we found that IL-6 modifies Lp(a)-associated cardiovascular risk.

Conclusión: GenProbCAD, a novel integrated genetic risk tool combining monogenic PVs, polygenic risk scores for CAD, and PRSLp(a), improves identification of individuals at high genetic risk for CAD across diverse populations.

Conclusión: In this primary prevention cohort, elevated Lp(a) levels were not associated with significant structural damage to the heart, carotid arteries, or increased aortic stiffness and were not associated with CV events and all-cause mortality.

Conclusión: OSA was associated with a continuously increased cardiovascular risk and a higher prevalence of HRP features as Lp(a) levels rose. Lp(a) may help identify ACS patients at higher cardiovascular risk, in whom the efficacy of OSA treatment should be further investigated.

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